By Sanjay Ghosh
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21 The mutations in FGFR are of gain-of-function type and increase the FGF binding capacity. Peroxisome Proliferator-activated Receptor (PPAR), Sebaceous Glands, Acne The PPAR subfamily consists of PPAR-α, PPAR- b/d, and PPAR-g. These three isotypes exhibit distinct patterns of tissue distribution and are involved in multiple distinctive but often complimentary physiological functions. 22 Similar observation was made by Downie et al. 23 Based on the cells used for studies in vitro, the role of PPAR activators on intracellular lipid production in the sebocytes has been conflicting.
Hodi FS, Dranoff G. Combinatorial cancer immunotherapy. Adv Immunol. 2006; 90:341–68. 27 28 Recent Advances in Dermatology 133. Krieg AM, et al. Therapeutic potential of Toll-like receptor 9 activation. Nat Rev. Drug Discov. 2006;5:471–84. 134. Tomai MA, Miller RL, Lipson KE, et al. Immune response modifiers: Imiquimod and future drugs for modulating the immune response. Drug Discov Today Ther Strateg [online]. 2006;3:342–52. 135. Lee J, et al. Molecular basis for the immunostimulatory activity of guanine nucleoside analogs: activation of Toll-like receptor 7.
J Invest Dermatol. 2000;114:706–11. 88. Wanke I, Steffen H, Christ C, et al. Skin commensals amplify the innate immune response to pathogen by activation of distinct signalling pathways. J Invest Dermatol. 2011;131:382–90. 89. Menzies BE, Kenoyer A. Signal transduction and nuclear responses in Staphylococcus aureus-induced expression of human bet-defensin 3 in skin keratinocytes. Infect Immun. 2006:74:6847–54. 90. Gallo RL, Nakatsuji T. Microbial symbiosis with the Innate Immune Defense System of the Skin.