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New York: Parthenon, 1996:89–93. Dalakas MC. Mechanism of action of intravenous immunoglobulin and therapeutic considerations in the treatment of autoimmune neurologic diseases. Neurology 1998; 51 (Suppl 5): S2–S8. Dalakas MC. Intravenous immunoglobulin in the treatment of autoimmune neuromuscular diseases: present status and practical therapeutic guidelines. Muscle Nerve 1999; 22: 1479–97. 3 Chronic urticaria Brigid F O’Donnell ANCA, antineutrophil immunoglobulins cytoplasmic antibody; IVIg, intravenous Introduction Urticaria, derived from the Latin urere, to burn, describes an eruption of transient, circumscribed cutaneous swellings.
In: Peter JB, Shoenfeld Y, eds. Autoantibodies. Amsterdam: Elsevier 1996; xxvii-xxix. O’Donnell BF, O’Neill CM, Francis DM et al. Human leucocyte antigen class II associations in chronic idiopathic urticaria. Br J Dermatol 1999; 140:853–8 . Niimi N, Francis DM, Kermani F et al. Dermal mast activation by autoantibodies against the high affinity IgE receptor in chronic urticaria. J Invest Dermatol 1996; 106: 1001–6. Grattan CEH, Boon AP, Eady RAJ et al. The pathology of the autologous serum skin test response in chronic urticaria resembles IgE-mediated late-phase reactions.
Down regulation of TGF-κ 1, mRNA and protein in the muscle of patients with inflammatory myopathy after treatment with high-dose intravenous immunoglobulin. Clin Immunol 2000; 94: 99–104. Dalakas MC. Experience with IVIg in the treatment of inflammatory myopathies. In: Kazatchkine L, Morel M, eds. Intravenous Immunoglobulin Research and Therapy. New York: Parthenon, 1996:275–84. Basta M, Dalakas MC. High-dose intravenous immunoglobulin exerts its beneficial effect in patients with dermatomyositis by blocking endomysial deposition of activated complement fragments.