By Prat A. (ed.)
This reference analyzes the mobile and molecular biology and mechanisms of the blood-brain barrier (BBB) and offers the latest experiences at the position of the BBB within the improvement and initiation of quite a lot of physiological and pathological stipulations affecting the imperative frightened method.
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Additional resources for The Blood-Brain Barrier and Its Microenvironment: Basic Physiology to Neurological Disease
Example text
This favors the nuclear translocation of b-catenin and its interaction with TCF/Lef transcription factors, possibly activating the transcription of TJ-genes like claudin-1. 16 Liebner and Engelhardt which is an inversion of the scheme known from TJ-development (33) as suggested by a shift from P-face to E-face associated TJs (28). Furthermore, the exctracellular matrix might contribute to BBB maintenance. In particular, agrin, a heparan sulfate proteoglycan, was found in the basement membranes of vessels with special barrier properties like those found in the brain, and was proposed to be involved in the development of the BBB (150).
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133. Gerhardt H, Wolburg H, Redies C. N-cadherin mediates pericytic–endothelial interaction during brain angiogenesis in the chicken. Dev Dyn 2000; 218:472–479. 134. Gerhardt H, Liebner S, Redies C, Wolburg H. N-cadherin expression in endothelial cells during early angiogenesis in the eye and brain of the chicken: relation to blood–retina and blood–brain barrier development. Eur J Neurosci 1999; 11:1191–1201. 135. Papkoff J, Brown AM, Varmus HE. The int-1 proto-oncogene products are glycoproteins that appear to enter the secretory pathway.