By Richard A. Bryant
Acute rigidity affliction: A instruction manual of concept, evaluation, and remedy is the 1st finished medical textual content on Acute rigidity affliction (AS D) considering its 1994 creation into DSM-IV as a diagnostic type. A uthors Richard A. Bryant and Allison G. Harvey define the reason a nd strategies to avoid the advance of PTSD by means of choosing and tr consuming people with ASD. Drawing from their pioneering scientific and rese arch event, they evaluation the underlying theoretical matters, then p resent a step by step advisor to assessing and treating ASD, and aspect the approaches for utilizing cognitive habit treatment to regard ASD. The authoritative e-book is a must-read for tutorial and scientific psychologi sts, psychiatrists, and different psychological healthiness execs operating with sufferers of trauma.
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Extra info for Acute Stress Disorder: A Handbook of Theory, Assessment, and Treatment
Example text
The decision to not stipulate that reexperiencing symptoms be distressing is inconsistent also with the finding that the sense of reliving the experience (which involves a strong adverse emotional response) is the most predictive acute symptom of chronic PTSD (Harvey & Bryant, 1998d). Moreover, acutely traumatized individuals experience varying degrees of distress associated with their intrusions (Ehlers & Steil, 1995). In one study, it was found that although the frequency of intrusions was comparable across terrorist attack survivors within the 1st week, there were variable levels of distress associated with 46 ACUTE STRESS DISORDER these memories (Shalev, Schreiber, & Galai, 1993).
BIOLOGICAL PROCESSES In recent years, there has been increasing attention to biological processes that can mediate acute stress reactions. A major line of research has implicated elevated arousal in the acute phase as a primary mediator of subsequent PTSD. , 1998). Similarly, we found that heart rates obtained before people were discharged from a hospital after a motor vehicle accident were significantly higher in those who later developed PTSD than those who did not (Bryant, Harvey, Guthrie, & Moulds, in press).
This activity will result in dampened arousal in the acute phase, which will be observed in lower heart rates and possibly will be mediated by reduced cortisol levels. In contrast, those who do not have dissociative tendencies will use more effortful coping skills, and these individuals will experience heightened arousal that will contribute to activation of the fear network. This aspect of the model accounts for the different arousal patterns in acutely traumatized people who dissociate and those who do not have this tendency.