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Extra resources for Nature outlook: Nutrigenomics. How food shapes health and evolution
Example text
CLOCK genotype frequencies did not deviate from the Hardy-Weinberg equilibrium. As SNPs, rs3749474, rs1464490 and rs4864548, were almost in complete LD and displayed a similar pattern of phenotypic associations, only the results for rs3749474 are presented. We first examined the association between the CLOCK SNPs and obesity parameters (Figure 2). We did not detect sex heterogeneity for any of the SNPs examined. Therefore, we present the results for men and women combined. We found significant associations with weight and BMI for SNPs rs3749474 and rs1801260 and with waist for rs3749474.
All of the anthropometric variables were significantly improved after treatment. Total cholesterol, uric acid and blood pressure were also significantly reduced after treatment. The mean duration of the treatment was 26±17 weeks. 85 kg. 24 and the rate of weight loss was 450 g per week. Genetic variation at the CLOCK rs1801260 SNP was associated with differences in body weight reduction. 005 Abbreviations: BMI, body mass index; WHR: waist–hip ratio. Data are presented as mean±EEM. 05 considered significant.
8 Fuller PM, Lu J, Saper CB. Differential rescue of light- and foodentrainable circadian rhythms. Science 2008; 230: 1074–1077. 9 Turek FW, Joshu C, Kohsaka A. Obesity and metabolic syndrome in circadian Clock mutant mice. Science 2005; 308: 1043–1045. 10 Rudic RD, McNamara P, Curtis AM. BMAL1 and CLOCK, two essential components of the circadian clock, are involved in glucose homeostasis. PLOS Biol 2006; 2: e377. 11 Scott EM, Carter AM, Grant PJ. Association between polymorphisms in the Clock gene, obesity and the metabolic syndrome in man.